High prevalence of patellar and achilles tendinopathies in futsal athletese.

Futsal (indoor version of soccer), in the last decade, has gained popularity all over the world. The game is played 5 – a – side, and during the competitions unlimited substitutions are permitted. Body movements, during playing, are characterized by frequent accelerating and stopping, jumping and tackling (Junge et al., 2004; Nogueira Ribeiro and Pena Costa, 2006). Consequently, physical demands are very high (Castagna et al., 2009). Epidemiological studies report a high incidence of knee and ankle injuries, owing to playing characteristics and field hard surface (Junge et al., 2004; Nogueira Ribeiro and Pena Costa, 2006). However, to our knowledge, systematic studies on the structural damage of Patellar (PT) and Achilles (AT) tendons have never been performed. Aim of this letter is to report the prevalence and incidence of PT and AT tendinopathies, in the players of a top level futsal team. Ultrasound (US) examinations were carried out at the beginning and at the end of the regular season. The diagnosis of tendinopathy and peritendinitis was performed using strict US criteria (Salini and Abate, 2011). Neovascularization was estimated by means of Colour Doppler, and graded as (0), (1+), (2++), (3+++), (4++++), according to the appearance of vessels inside the tendon (Alfredson and Ohberg, 2005). Demographic and anthropometric data were registered. By self report, information about injuries, suffered in the past, or during the season, were collected. An injury was defined as pain or tendon discomfort regardless of the subsequent absence from match or training. Players who had tendon damage at US evaluation were compared with those free from abnormalities. Data are reported as mean and standard deviation, frequencies and percentage. Student's t and χ 2 tests were used. At the beginning of the season, 7 players (38.8 %) showed tendon degeneration, associated with neovascularisation in 7 / 9 tendons (Table 1). Out of them, all asymptomatic when observed, 5 had complained injuries in the past. The remaining 11 athletes were asymptomatic and without tendon abnormalities. At the end of the season, in 3 players, among the ones who did not show tendon damage at the beginning, US revealed the onset of peritendinitis with neovascularisation. In most cases, tendinopathy was observed in the non dominant leg. Therefore, a total of 10 athletes (55.5 %) showed tendon damage at the end of the season. To sum up, 12 tendons had US signs of tendinopathy associated with neovascularisation in 10 (Table 1). Among these players, 8 reported tendon discomfort during regular season, and 4 were forced to stop playing for a short period (~ 10 days). All the US positive subjects had played for several years and were older (Table 2). These results show that PT and AT tendinopathies are very frequent among futsal players. The high prevalence of tendon lesions, due to acute traumas and, more frequently, to overuse degeneration, may be explained by the playing characteristics : the frequent accelerating and stopping, jumping, tackling, and abrupt jerking movements can indeed cause significant stress on knee and ankle (Junge et al., 2004, Nogueira Ribeiro and Pena Costa, 2006). Moreover, the low heel footwear can expose AT to higher straining and stretching, and the hard and adherent playing surface, that does not cushion the blows of the heel, can further stress this tendon (Cain et al., 2007). The higher prevalence of lesions in the non dominant leg can be explained by the fact that periods of play are mostly spent in single – leg stance on this leg, while the dominant foot manipulates the ball (Cain et al., 2007). To our knowledge, this is the first US study of tendinopathies in futsal, and, therefore, every comparison with other sports may be misleading. For example, Fredberg and Bolvig (Fredberg and Bolvig, 2002) found, in asymptomatic